HBOT Australia | Education

HBOT Australia

Phone: +61 427 480 897

Add/Update information

Reviews

Tags

• Other
• Brand
• Education
• Website
• Personal blog
• Editorial/Opinion

24.01.2022 Can You Heal from TBI and CTE https://tbitherapy.com/cte-tbi-treatment/ Summary for quick read or full article and references below... Summary Rest, heal, treatment,oxygen, nutrients....or it dies For every TBI or CTE patient with or without symptoms, at a cellular level, they are deficient in the resources, oxygen and nutrients, to provide enough energy to function as well as to heal the brain. Hence, it makes since that providing oxygen, through pressure hyperbaric oxygen therapy, and nourishment from autologous plasma dripped directly into the brain through the upper part of the nose can help traumatic brain injury patients as well as potentially prevent CTE. However, once a TBI patient has progressed to the degenerative state of CTE, it can be much harder to restore the neurons to a more functional state. By Dr. John Hughes, DO As a TBI treatment specialist, I see a variety of patients with traumatic brain injuries (TBI) from military blast events, professional and high school sports, motor vehicle accidents, blunt traumas, or other causes. These patients almost always have some degree of mild to severe chronic post-concussive symptoms including mental fatigue, loss of concentration, memory loss, loss of decision-making, loss of motivation, insomnia, loss of libido, depression, anger, and even suicide ideation. These patients come to my clinic, TBI Therapy, with numerous questions about whether they can actually heal from their traumatic brain injury and if it may eventually progress to CTE in their later years. What is CTE? Boston University of Medicine researchers define it: Chronic Traumatic Encephalopathy (CTE) is a progressive degenerative disease of the brain found in people with a history of repetitive brain trauma (often athletes) The repeated brain trauma triggers progressive degeneration of the brain tissue, including the build-up of an abnormal protein called tau. These changes in the brain can begin months, years, or even decades after the last brain trauma or end of active athletic involvement (Boston University Research). Ultimately, if TBI patients do not receive effective treatment, their TBI may eventually progress to become CTE in which their post-concussive symptoms actually become more permanent and pronounced, primarily, per the current medical theory, due to the build of tau proteins. What are Tau Proteins? Tau proteins are intracellular proteins of neurons that support microtubules, hollow fibrous tubes that give structure to the cell. Tau proteins in proper numbers are essential and normal in everyone’s brain. However, when patients have untreated or poorly treated concussions or more severe TBIs, these proteins tend to hyperaccumulate in the cells and block neuronal signaling, particularly along axonal pathways, leading the symptoms associated with CTE, Alzheimer’s disease, and Parkinson’s. When these Tau proteins hyperaccumulate in cells, they begin to act a lot like scar tissue that occurs after sports injuries or surgeries in skin, ligaments, tendons, and even joints. The scar tissue inside tendons and ligaments is also fibrous tissue called fibrosis. When viewed under a microscope, the fibrotic scar tissue looks a lot like disorganized calcified spider webs in comparison to the normal longitudinal patterns of collagen healthy tendons and ligaments. This scar tissue, operating in a similar way to Tau proteins, hyper-accumulates and ultimately ends up trapping nerve fibers and blocking nerve transmission. Treatment for this fibrosis in tendons and ligaments requires an injection of autologous human plasma (aka platelet rich plasma from the same patient), which is rich in growth factors as well as anti-fibrotic factors, and oxygen to help restore the tissue to a healthy state. Tau immunostained sections of medial temporal lobe from 3 individuals: The Tau stains are brown. It is theorized that treating TBI patients with oxygen, including hyperbaric oxygen therapy (HBOT), and plasma rich in growth and anti-fibrotic factors may also prevent Tau protein accumulation associated with CTE. In the same way that scar tissue forms in tissues due to lack of oxygen and nourishment, this rationale explains why Tau proteins develop in the neurons of the brain. For every TBI or CTE patient with or without symptoms, at a cellular level, they are deficient in the resources, oxygen and nutrients, to provide enough energy to function as well as to heal the brain. Hence, it makes since that providing oxygen, through pressure hyperbaric oxygen therapy, and nourishment from autologous plasma dripped directly into the brain through the upper part of the nose can help traumatic brain injury patients as well as potentially prevent CTE. However, once a TBI patient has progressed to the degenerative state of CTE, it can be much harder to restore the neurons to a more functional state. References Boston University Research: CTE Center. Retrieved January 15, 2020 from http://www.bu.edu/cte/about/frequently-asked-questions/. Boussi-Gross, R., Golan, H., Fishlev, G., Bechor, Y., Volkov, O., et al. (2013) Hyperbaric Oxygen Therapy Can Improve Post Concussion Syndrome Years after Mild Traumatic Brain Injury Randomized Prospective Trial. PLoS ONE 8(11): e79995. doi: 10.1371/journal.pone.0079995. Cantu, R. (August, 2013). What Physical and Cognitive Rest Really Mean After a Concussion. Retrieved from https://www.brainline.org//what-physical-and-cognitive-res. Donahue, D. L., Beck, J., Fritz, B., Davis, P., Sandoval-Cooper, M., Thomas, S. G., . . . Castellino, F. J. (2014). Early platelet dysfunction in a rodent model of blunt traumatic brain injury reflects the acute traumatic coagulopathy found in humans. Journal of Neurotrauma, 31(4), 404-410. doi: 10.1089/neu.2013.3089 Eve, D. J., Steele, M. R., Sanberg, P. R., & Borlongan, C. V. (2016). Hyperbaric oxygen therapy as a potential treatment for post-traumatic stress disorder associated with traumatic brain injury. Neuropsychiatric disease and treatment, 12, 2689. Gladstone Institutes. (2008). Collagen May Help Protect Brain Against Alzheimer’s Disease. ScienceDaily. Retrieved August 15, 2016 from www.sciencedaily.com/releases/2008/12/081210150713.htm Hadanny, A., Abbott, S., Suzin, G., Bechor, Y., & Efrati, S. (2018). Effect of hyperbaric oxygen therapy on chronic neurocognitive deficits of post-traumatic brain injury patients: retrospective analysis. BMJ open, 8(9), e023387. Hanson, L. R., & Frey, W. H. (2008). Intranasal delivery bypasses the blood-brain barrier to target therapeutic agents to the central nervous system and treat neurodegenerative disease. BMC neuroscience, 9(Suppl 3), S5. Harch, P. G., Andrews, S. R., Fogarty, E. F., Lucarini, J., & Van Meter, K. W. (2017). Case control study: hyperbaric oxygen treatment of mild traumatic brain injury persistent post-concussion syndrome and post-traumatic stress disorder. Medical Gas Research, 7(3), 156174. http://doi.org/10.4103/2045-9912.215745 Harch, P. G., Andrews, S. R., Fogarty, E. F., Amen, D., Pezzullo, J. C., Lucarini, J., & Van Meter, K. W. (2012). A phase I study of low-pressure hyperbaric oxygen therapy for blast-induced post-concussion syndrome and post-traumatic stress disorder. Journal of neurotrauma, 29(1), 168-185. Hardy, P., Johnston, K. M., De Beaumont, L., Montgomery, D. L., Lecomte, J. M., Soucy, J. P., & Lassonde, M. (2007). Pilot case study of the therapeutic potential of hyperbaric oxygen therapy on chronic brain injury.Journal of the neurological sciences, 253(1), 94-105. Hu, Q., Manaenko, A., Xu, T., Guo, Z., Tang, J., & Zhang, J. H. (2016). Hyperbaric oxygen therapy for traumatic brain injury: bench-to-bedside. Medical Gas Research, 6(2), 102110. http://doi.org/10.4103/2045-9912.184720 Hughes, J. (2018). Case Report for a mTBI Patient Treated with Intermittent Home HBOT, Intranasal and IV PRP Cocktail, Intranasal Pluripotent Stem Cells from Peripheral Blood, Intranasal Insulin, Cranial Osteopathy, and a Ketogenic Diet. Hughes, J. (2016). Case Report for TBI (Traumatic Brain Injury) Patient Treated with A Protocol of HBOT (Hyperbaric Oxygen Therapy), Autologous Human Plasma, Cranial Therapy, EEG Biofeedback, IV Nutrition, and Adult Stem Cells. Huang, L., & Obenaus, A. (2011). Hyperbaric oxygen therapy for traumatic brain injury. Medical Gas Research, 1, 21. http://doi.org/10.1186/2045-9912-1-21. Matsui M and Y Tabata. 2012. Enhanced angiogenesis by multiple release of platelet-rich plasma contents and basic fibroblast growth factor from gelatin hydrogels. Acta Biomater. 8(5):1792-801. doi: 10.1016/j.actbio.2012.01.016 Middleton KK et al., 2012. Evaluation of the effects of platelet-rich plasma (PRP) therapy involved in the healing of sports-related soft tissue injuries. Iowa Orthop J. 32:150-63. Stoller, K. P. (2011). # Hyperbaric oxygen therapy (1.5 ATA) in treating sports related TBI/CTE: two case reports. Medical gas research, 1(1), 1-6. Tal, S., Hadanny, A., Sasson, E., Suzin, G., & Efrati, S. (2017). Hyperbaric oxygen therapy can induce angiogenesis and regeneration of nerve fibers in traumatic brain injury patients. Frontiers in Human Neuroscience, 11, 508. Tal, S., Hadanny, A., Berkovitz, N., Sasson, E., Ben-Jacob, E., & Efrati, S. (2015). Hyperbaric oxygen may induce angiogenesis in patients suffering from prolonged post-concussion syndrome due to traumatic brain injury. Restorative Neurology and Neuroscience. 33(6):943-51. doi: 10.3233/RNN-150585. Tau Proteins. Retrieved January 15, 2020 from https://en.wikipedia.org/wiki/Tau_protein The Function Of Microtubules. Retrieved January 15, 2020 from http://www.softschools.com///the_function_of_microtubules/ Thom, S. R., Bhopale, V. M., Velazquez, O. C., Goldstein, L. J., Thom, L. H., & Buerk, D. G. (2006). Stem cell mobilization by hyperbaric oxygen. American Journal of Physiology-Heart and Circulatory Physiology, 290(4), H1378-H1386. Weil, L., Kol, C. P., Sul, K., & Chiu, W. T. (2008). Effect of hyperbaric oxygen on patients with traumatic brain injury. Acta Neurochirurgica Supplements Editor: H.-J. Steiger, 101, 145-149. Wolf, G., Cifu, D., Baugh, L., Carne, W., & Profenna, L. (2012). The effect of hyperbaric oxygen on symptoms after mild traumatic brain injury. Journal of neurotrauma, 29(17), 2606-2612. Wright, J. K., Zant, E., Groom, K., Schlegel, R. E., & Gilliland, K. (2009). Case report: treatment of mild traumatic brain injury with hyperbaric oxygen. Zhang, Y., Ying, G., Ren, C., Jizhang, Y., Brogan, D., Liu, Z., . . . Ji, X. (2015). Administration of human platelet-rich plasma reduces infarction volume and improves motor function in adult rats with focal ischemic stroke. Brain Research, 1594, 267-273. Zhang, J., Jiang, R., Liu, L., Watkins, T., Zhang, F., & Dong, J. (2012). Traumatic brain injury-associated coagulopathy. Journal of Neurotrauma, 29(17), 2597-2605. doi: 10.1089/neu.2012.2348

20.01.2022 *US VETERANS ONLY FREE TREATMENT BELOW * AUSTRALIA VETERANS - YOUR COUNTRY IS TURNING ITS BACK We (MY WIFE AND I ) are fighting here for your right with DVA, to receive the best treatments available,we have show cased and created a precedent in the government, but implementation of treatment for veterans, the public , everyone is difficult and long with minimal support. We have political and public and medical support, but we need YOUR HELP. ... NO chambers means no treatment FOR EVERYONE There are over 4400 External Support organisations, for veterans, they do a phenomenal job, but no support. The US have 1200 private centres and public government approval , we in Australia have 3-5 private centres and public hospital in each capital city. For a country the size of the US, its disgraceful. ESOs where are you, help us ..... we have the facilities at PRO PATRIA Wagga ,to make this a vailable to 10000s, ...... Help us ..... be part of leading the change

16.01.2022 ADMIN is back fully and beaten COVID-19 (HBOT is why)

14.01.2022 Note I had covid so I have the right to talk about it. https://www.futurity.org/silent-hypoxia-covid-19-lungs-oxy/

14.01.2022 The coverup and denial of treatment The problem is not whether hyperbaric oxygen is effective for treating brain injuries, but why the interference in offering this therapy to those who need it. The problem is not the treatment, the problem is the government and big pharma.... Note this is 2014 US article. Everything has now been clinically proven with 100s of studies. The article shows what is happening here in AUSTRALIA. CORRUPTION, CRIME, DECEPTION TO CONTROL A INDUSTRY, AS ITS NOT IN A PILL Abstract Background The modern age of hyperbaric medicine began in 1937; however, today few know about hyperbaric oxygen’s effects on the body and medical conditions outside of diving medicine and wound care centers - a serious ethical issue as there are 20 US military veterans committing suicide every day directly related to Traumatic Brain Injury/Post Traumatic Stress Disorder. The problem is not whether hyperbaric oxygen is effective for treating brain injuries, but why the interference in offering this therapy to those who need it. Discussion Up against black-boxed anti-depressants that are not efficacious, it should be a no-brainer to use a safe, off-label drug, but in the case of military veterans, every suicide might be seen as a tremendous cost saving to certain technocrats. The unspoken rationale is that if the military were to embrace hyperbaric oxygen as the efficacious therapy that it is then current active troops that have suffered injuries will come forward and seek treatment and benefits for their Traumatic Brain Injuries now that they know there is a viable therapy and in so doing troop strength will be decimated. So, to attempt to delay the acceptance of hyperbaric oxygen the Department of Defense has funded faux-studies claiming low pressure room air to be a placebo or sham, and then proclaiming there is no statistical difference between treatment arms and sham or placebo treatment arms. With few who understand hyperbaric medicine there is almost no one to call them on this subterfuge and prevarication. Many peer-reviewed articles have been published in the last decade that demonstrate hyperbaric oxygen is effective in repairing an injured brain even long after that injury took place. One of the most notable showed that blast-induced brain injured war veterans experienced a 15 point IQ increase (p < 0.001). Summary Hyperbaric oxygen is an efficacious, benign and humanitarian way to affect brain repair but it has not been adopted because it lacks patent protection and has no large corporate sponsors. It has also met interference because other agendas are present be they the protection of the status quo, myopic budgetary constraints, or perceived liability issues. https://medicalgasresearch.biomedcentral.com//s13618-015-0

02.01.2022 Enough is enough.....we need this not only for veterans but for everyone. As the results and applications from it flow onto the public sector and your rights.